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Liraglutide can enter specific brain regions associated with appetite regulation, electrophysiological measurements of mouse brain sections showed that GLP-1 directly stimulated promelanocortin (POMC)/cocaine and amphetamine regulated transcript (CART) neurons and indirectly inhibited neurotransmission of neuropeptide Y (NPY) and agouti-related peptide (AgRP) in arcuate nucleus neurons through GABA-dependent signal transduction

While traditionally recognized for its role in glucose homeostasis and insulin secretion, GLP-1 exerts significant effects on the central nervous system through activation of GLP-1 receptors (GLP-1R) distributed throughout various brain regions

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This combination can create a more significant calorie deficit than using either medication alone